Database : HANSEN Search on : CORPOS DE HEINZ [Subject descriptor] References found : 3 [refine] Displaying: 1 .. 3   in format [Detailed]

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Id: 17899 Author: Rimoli, Luiz Fernando; Godoy, Moacir Fernandes de. Title: Quantificacao do estresse oxidativo no sangue de hansenianos sob o efeito ou nao de tratamento especifico / Quantification of the oxidative stress in the blood of people with Hansen's disease undertaking or not a specific treatment Source: Hansen. int;26(2):93-98, jul.-dez. 2001. ilus, tab. Abstract: Neste estudo avaliamos a quantificaçäo do estresse oxidativo no sangue de hasenianos sob efeito ou näo de tratamento específico. Foram coletadas amostras de sangue de 62 portadores de hanseníase e 13 sadios. Destes 62 pacientes, 35 eram de forma multibacilar e faziam tratamento com clofazimina, dapsona e rifampicina; 16 de forma paucibacilar tratados com dapsona e rifampicina; 11 dos pacientes estavam sem tratamento, sendo 5 da forma multibacilar e 6 da forma paucibacilar. As amostras foram levadas para análises laboratoriais onde foram feitas as dosagens de metahemoglobina e contagem de corpos de Heinz. Os resultados encontrados foram submetidos à análise estatística com cálculo da Odds Ratio e Intervalo de Confiança 95 por cento. Concluimos que a doença haseníase näo foi a causadora do estresse oxidativo, e sim a terapêutica administrada, e o grupo que fazia uso da clofazimina apresentou piores resultados, ou seja, maior estresse oxidativo.(AU). Descriptors: ESTRESSE OXIDATIVO/imunol ESTRESSE OXIDATIVO/fisiol HANSENIASE/compl HANSENIASE/diag HANSENIASE/psicol CORPOS DE HEINZ/patol CORPOS DE HEINZ/fisiol Limits: ESTUDO COMPARATIVO HUMANO MASCULINO FEMININO Electronic Medium: http://hansen.bvs.ilsl.br/textoc/hansenint/v21aov29/2001/PDF/v26n2/v26n2a01.pdf / pt Location: BR191.1

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Id: 13277 Author: Rasbridge, Marian R; Scott, G. L Title: The haemolytic action of dapsone: the effect on red-cell glycolysis ..- Source: s.l; s.n; feb. 1973. 13 p. tab, graf. Abstract: Some aspects of red-cell matabolism were studied in blood samples taken from patients on long-term dapsone therapy. Glucose consumption by the Embden-Meyerhof pathway (EMP) was not abnormal and adenosine-triphosphate (ATP) levels lay within the normal range and were well maintained during incubation. Hexose monophosphate patchway (HMP) activity was increased above that which would be expected from the age of the red-cell population. Red-cell reduced glutathione (GSH) levels tended to be lower than normal, proportional to the dose od dapsone and GSH levels were unstable on incubation. Red-cell fractionation studies showed that the older cell fraction had lower GSH levels and more Heinz bodies but proportionally greater HMP activity than the younger cell fraction. It is suggested that the low GSH levels are probably due to the binding of GSH to sulphydryl groups in haemoglobin and possibly the red-cell membrane. The increased HMP activity in the older cells many, in part, be a compensatory mechanism.(AU). Descriptors: ADENOSINA TRIFOSFATO/sangue DAPSONA/ef adv DAPSONA/farmacol DERMATITE HERPETIFORME/quimioter ENVELHECIMENTO ERITROCITICO ERITROCITOS/enzimol ERITROCITOS/metab GLUCOSEFOSFATO DESIDROGENASE/sangue GLUTATIONA/sangue CORPOS DE HEINZ Electronic Medium: http://www.ilsl.br Location: BR191.1; 01856/s

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Id: 9502 Author: Chan, Heng-Leong; Lee, Kok-Onn Title: Tonsillar membrane in the DDS (dapsone) syndrome ..- Source: s.l; s.n; 1991. 2 p. ilus. Descriptors: DAPSONA ERUPÇAO POR DROGA CORPOS DE HEINZ HANSENIASE TUBERCULOIDE DOENÇAS FARINGEAS DOENÇAS FARINGEAS SINDROME TONSILA TONSILA Limits: RELATO DE CASO Location: BR191.1; 06358/s

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